2 edition of Evaluation of the relationships between cadmium exposure and indicators of kidney function found in the catalog.
Evaluation of the relationships between cadmium exposure and indicators of kidney function
by (Monitoring and Assessment Research Centre, Chelsea College, University of London) in (London)
Written in English
Bibliography, p45-46. -Includes index.
|Statement||by Malcolm Hutton ; a technical report (1983) prepared by Monitoring and Assessment Research Centre, Chelsea College, University of London for the Commission of the European Communities.|
|Series||MARC report -- no.29|
|Contributions||Chelsea College. Monitoring and Assessment Research Centre., Commission of the European Communities.|
|The Physical Object|
|Number of Pages||46|
To evaluate the potential relationship between arsenic and CKD, we conducted a systematic review of epidemiologic studies that have investigated the association between inorganic arsenic exposure, assessed via geographical measures (e.g., living in a high exposure area), environmental markers (e.g., arsenic in drinking water) or biomarkers (e.g., urine arsenic), and CKD Cited by: Evaluation for chronic cadmium exposure may also include a complete blood count to check for anemia and liver function tests. In cases with renal complications, a set of skeletal X-rays should be ordered to check for bone-related complications of cadmium exposure such as osteomalacia, osteopenia, and skeletal fractures.
The increasing environmental and occupational exposure of populations to cadmium creates the need for biological indicators of cadmium exposure and toxicity. The advantages and disadvantages of monitoring blood cadmium, urinary, fecal, hair, and tissue cadmium, serum creatinine, β2-microglobulin, α1-antitrypsin and other proteins, and urinary amino acids, enzymes, total proteins, Cited by: Journal Article: Evaluation of biological indicators of body burden of cadmium in humans.
As expected, diabetes affected the kidney function, although only in insulin-dependent women, of whom about half had type II diabetes. More important, we found an interaction between cadmium and diabetes, as suggested in previous studies (Buchet et al. ). Hence, the lowest observed effect level is expected to be lower in diabetics but could Cited by: indicator of changes in kidney function suggesting possible kidney damage later in life. Within task (Deliverable ), evaluation of fractions of urinary samples exceeding the threshold value of 1 μg/g creatinine has been assessed for the available HBM data for .
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Hutton M () Evaluation of the relationships between cadmium exposure and indicators of kidney function. London Chelsea College, University of London, 46 pp (MARC Report No 29) Google Scholar IARC (a) IARC by: 1.
Cadmium (Cd) is a toxic metal. This study was aimed to estimate the potential health risks in a Cd-polluted district in China, and examine the relationship between urinary cadmium(UCd) and hypertension and impaired kidney function at low exposure levels (UCd: GM μg/g creatinine).Cited by: In spite of low-level cadmium exposure, the present study found a significant association between kidney cadmium and excretion of a low molecular weight protein in healthy kidney donors.
This indicates that an effect on renal tubules may still be an early effect of cadmium by: The first study of the relation between Cd in kidney, blood and urine at low U-Cd Simultaneous samples were collected from healthy kidney donors. There was a nonlinear relationship between cadmium in kidney and urine.
Estimates of the kidney cadmium half-time were 18–44 years, depending on model used. Previous data seem to underestimate Cited by: Introduction: Cadmium (Cd) is toxic to the kidney and a major part of the body burden occurs here.
Cd in urine (U-Cd) and blood (B-Cd) are widely-used biomarkers for assessing Cd exposure or body. Results of both human and animal studies suggest an association between Cd exposure, elevated blood glucose levels and the development of diabetes and diabetes-related kidney disease.
In addition, many epidemiological and experimental studies suggest that Cd potentiates or exacerbates diabetic by: Introduction: Cadmium (Cd) is toxic to the kidney and a major part of the body burden occurs here.
Cd in urine (U-Cd) and blood (B-Cd) are widely-used biomarkers for assessing Cd exposure or body burden. However, empirical general population data on the relationship between Cd in kidney (K-Cd. A relationship between smoking and kidney disease has been suggested (Bleyer et al.,Fox et al., ), with evidence indicating that exposure to cadmium (Richter et al., ) and other toxicants in cigarettes such as nicotine (Jain and Jaimes, ) may have deleterious effects on the by: 6.
A survey regarding environmental exposure to cadmium that was conducted in Belgium (Cadmibel, –) showed an association between cadmium exposure and an increased prevalence of kidney dysfunction (above the 95th percentile in the control group) (Buchet et al., ).
The results of several studies performed on populations that were environmentally exposed to cadmium indicate that changes in sensitive renal biomarkers may occur at urinary cadmium Cited by: By estimating dietary cadmium, we are able to assess exposure in a manner that avoids any influence of kidney function on the exposure metric.
In addition, dietary cadmium exposure allows for the impact of cadmium in food to be assessed independently of cadmium from other sources such as smoking. Methods Study populationsCited by: L. FRIBERG Cadmiumin liverjug/N wetweight F F ~ J SUI *0 Groups with normal:5 People with-excessive-exposure cadmium exposure t at age _ + proteinuria S-Sweden U-USA _ _ severe J-Japan morphological (mean-1 S.D.) changes FIGURE 2.
Renal cortex cadmium concentrations as a func- tion ofrenal effects observed (62): (0) industrial exposure; (o) general environmental exposure. Cadmium (Cd) is a toxic metal. This study was aimed to estimate the potential health risks in a Cd-polluted district in China, and examine the relationship between urinary cadmium(UCd) and Cited by: Ferraro P, Costanzi S, Naticchia A, Sturniolo A and Gambaro G () Low level exposure to cadmium increases the risk of chronic kidney disease: analysis of the NHANESBMC Public Health, /,Online publication date: 1-DecCited by: This study was conducted to evaluate the toxic effects of cadmium (Cd) on the kidney function and bone development in laying hens.
A total of Hy-line laying hens aged 38 weeks were randomly. Many of these tubular and glomerular function indicators were significantly correlated with both cumulative exposure index and liver cadmium burden. Using cumulative exposure index and liver cadmium as estimates of dose, a two phase linear regression model was applied to identify an inflection point signifying a threshold level above which changes in renal function by: With constant exposure, it is obvious that a dose‐response relationship between the cadmium level in blood and the prevalence of signs of impaired kidney function can also be established.
A cadmium exposure of 1 µg/ ml whole blood in the group can be tolerated without kidney damage (Buchet et Author: G. Lehnert, H. Drexler, A. Hartwig. Evaluation of the relationships between cadmium exposure and indicators of kidney function / by Malcolm Microcirculation and tubular urine flow in the mammalian kidney cortex (in vivo microscopy) / M.
Steinha Renal function: physiological and medical aspects / John W. Bauman, Francis P. Chinard. Lead workers; cadmium exposure; kidney function; biological mechanism; cadmium; Weaver et al1 have published data on the associations between urinary cadmium and four glomerular filtration measures or N-acetyl-β-d-glucosaminidase (NAG) as a biological renal target subjects were lead-handling workers and they concluded a paradoxical result that urinary cadmium Cited by: 1.
Measurement of kidney function, Cd excretion rates and toxicity. and a linear relationship between β2-MG and albumin was evident in hypertensive subjects. In an Associations between cadmium exposure and circulating levels of sex hormones in postmenopausal by: 1.
The dose–response relationship between U-Cd levels and prevalence of U-NAG above the reference value support the need for measures to reduce environmental Cd exposure. Over the last century, emissions of cadmium from various industries and the combustion of waste and fossil fuels have resulted in a considerable elevation of the concentrations Cited by:.
Objectives The nephrotoxicity of cadmium at low levels of exposure, measured by urinary cadmium, has recently been questioned since co-excretion of cadmium and proteins may have causes other than cadmium toxicity. The aim of this study was to explore the relation between kidney function and low or moderate cadmium levels, measured directly in kidney biopsies.
Methods We analysed cadmium Cited by: Adequate data on cadmium exposures were available to allow evaluation of dose-response relationships between cadmium exposure and lung cancer.
A statistically significant excess of lung cancer attributed to cadmium exposure was observed in this study even when confounding variables such as co-exposure to arsenic and smoking habits were taken.Environmental exposure to cadmium may cause kidney damage and tubular proteinuria.
We investigated the relationship between low-level cadmium exposure and end-stage renal disease (ESRD), indicated by renal replacement therapy (RRT), in a Swedish population environmentally or occupationally exposed to cadmium.
Based on records of all persons in the population previously or Cited by: